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The “structurally minimal” isoform KChIP2d modulates recovery of K(v)4.3 N-terminal deletion mutant Δ2-39
Mechanisms underlying K(v)4 (Shal type) potassium channel macroscopic (open state) inactivation and recovery are unknown, as are mechanisms by which KChIP2 isoforms modulate these two processes. In a recent study (Xenopus oocytes, 2 microelectrode voltage clamp) we demonstrated that: i) Partial dele...
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| 主要な著者: | , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
Landes Bioscience
2011
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3225751/ https://ncbi.nlm.nih.gov/pubmed/21422811 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4161/chan.5.3.15313 |
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