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Involvement of Neuronal Nitric Oxide Synthase in Ongoing Fetal Brain Injury following Near-Term Rabbit Hypoxia-Ischemia

Neuronal nitric oxide synthase (nNOS) and nitric oxide (NO) are implicated in neuronal injury following acute hypoxia-ischemia (HI). Our hypothesis was that NO from nNOS is responsible for ongoing mitochondrial dysfunction in near-term fetal HI. Recently, we synthesized new selective nNOS inhibitors...

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Autors principals: Rao, Suma, Lin, Zhenlang, Drobyshevsky, Alexander, Chen, Lina, Ji, Xinhai, Ji, Haitao, Yang, Yirong, Yu, Lei, Derrick, Matthew, Silverman, Richard B., Tan, Sidhartha
Format: Artigo
Idioma:Inglês
Publicat: S. Karger AG 2011
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Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC3225249/
https://ncbi.nlm.nih.gov/pubmed/21757865
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1159/000327241
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