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Memory consolidation in both trace and delay fear conditioning is disrupted by intra-amygdala infusion of the protein synthesis inhibitor anisomycin

Memory for delay fear conditioning requires the synthesis of new mRNA and protein in the basolateral amygdala. It is currently unknown whether similar molecular processes in the amygdala are required for the formation of trace fear memory, in which a stimulus-free interval is inserted between the co...

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Main Authors: Kwapis, Janine L., Jarome, Timothy J., Schiff, Janet C., Helmstetter, Fred J.
פורמט: Artigo
שפה:Inglês
יצא לאור: Cold Spring Harbor Laboratory Press 2011
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גישה מקוונת:https://ncbi.nlm.nih.gov/pmc/articles/PMC3207254/
https://ncbi.nlm.nih.gov/pubmed/22028394
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1101/lm.023945.111
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