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Appetite for destruction: the inhibition of glycolysis as a therapy for tuberous sclerosis complex-related tumors
The elevated metabolic requirements of cancer cells reflect their rapid growth and proliferation and are met through mutations in oncogenes and tumor suppressor genes that reprogram cellular processes. For example, in tuberous sclerosis complex (TSC)-related tumors, the loss of TSC1/2 function cause...
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| Main Authors: | , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
BioMed Central
2011
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| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3198763/ https://ncbi.nlm.nih.gov/pubmed/22018140 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/1741-7007-9-69 |
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