PRENATAL ADMINISTRATION OF THE CYTOCHROME P4501A INDUCER, B-NAPHTHOFLAVONE (BNF), ATTENUATES HYPEROXIC LUNG INJURY IN NEWBORN MICE: IMPLICATIONS FOR BRONCHOPULMONARY DYSPLASIA (BPD) IN PREMATURE INFANTS

Lignende værker

• Omeprazole Attenuates Hyperoxic Lung Injury in Mice via Aryl Hydrocarbon Receptor Activation and Is Associated with Increased Expression of Cytochrome P4501A Enzymes
  af: Shivanna, Binoy, et al.
  Udgivet: (2011)
• Newborn Mice Lacking the Gene for Cyp1a1 Are More Susceptible to Oxygen-Mediated Lung Injury, and Are Rescued by Postnatal β-Naphthoflavone Administration: Implications for Bronchopulmonary Dysplasia in Premature Infants
  af: Maturu, Paramahamsa, et al.
  Udgivet: (2017)
• OXYGEN-MEDIATED LUNG INJURY IN MICE LACKING THE GENE FOR NRF2: RESCUE WITH THE CYTOCHROME P4501A-INDUCER, BETA-NAPHTHOFLAVONE (BNF), AND DIFFERENTIAL SEX-SPECIFIC EFFECTS
  af: Callaway, Danielle A., et al.
  Udgivet: (2020)
• β-Naphthoflavone treatment attenuates neonatal hyperoxic lung injury in wild type Cyp1a2-knockout mice
  af: Lingappan, Krithika, et al.
  Udgivet: (2017)
• Disruption of Cytochrome P4501A2 in mice leads to increased susceptibility to hyperoxic lung injury
  af: Wang, Lihua, et al.
  Udgivet: (2015)