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Impairment of Lon-Induced Protection Against the Accumulation of Oxidized Proteins in Senescent Wi-38 Fibroblasts

Oxidative damage to mitochondrial proteins is thought to contribute to the aging process, but the Lon protease normally degrades such proteins. In early-passage WI-38 human lung fibroblasts, Lon expression is rapidly induced during H(2)O(2) stress, which prevents the accumulation of oxidized protein...

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Bibliografiska uppgifter
Huvudupphovsmän: Ngo, Jenny K., Pomatto, Laura C. D., Bota, Daniela A., Koop, Alison L., Davies, Kelvin J. A.
Materialtyp: Artigo
Språk:Inglês
Publicerad: Oxford University Press 2011
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Länkar:https://ncbi.nlm.nih.gov/pmc/articles/PMC3193527/
https://ncbi.nlm.nih.gov/pubmed/21868393
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/gerona/glr145
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