Inhibition of c-Jun N-Terminal Kinase Attenuates Low Shear Stress–Induced Atherogenesis in Apolipoprotein E–Deficient Mice

Atherosclerosis begins as local inflammation of arterial walls at sites of disturbed flow, such as vessel curvatures and bifurcations with low shear stress. c-Jun NH(2)-terminal kinase (JNK) is a major regulator of flow-dependent gene expression in endothelial cells in atherosclerosis. However, litt...

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Main Authors: Wang, Juan, An, Feng Shuang, Zhang, Wei, Gong, Lei, Wei, Shu Jian, Qin, Wei Dong, Wang, Xu Ping, Zhao, Yu Xia, Zhang, Yun, Zhang, Cheng, Zhang, Ming-Xiang
Formato: Artigo
Idioma:Inglês
Publicado em: ScholarOne 2011
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Articles
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3188877/
https://ncbi.nlm.nih.gov/pubmed/21629969
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.2119/molmed.2011.00073
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