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Presenilin-2 modulation of ER-mitochondria interactions: FAD mutations, mechanisms and pathological consequences
Presenilin (PS) mutations are the main cause of Familial Alzheimer's Disease (FAD) and have been demonstrated to cause an imbalance of intracellular Ca(2+) homeostasis. Though PS1 and 2 are generally considered to behave similarly in terms of their effects on Ca(2+) handling, we have recently d...
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| Autors principals: | , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
Landes Bioscience
2011
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3187908/ https://ncbi.nlm.nih.gov/pubmed/21980580 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4161/cib.4.3.15160 |
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