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Presenilin 2 modulates endoplasmic reticulum (ER)–mitochondria interactions and Ca(2+) cross-talk
Presenilin mutations are the main cause of familial Alzheimer's disease (FAD). Presenilins also play a key role in Ca(2+) homeostasis, and their FAD-linked mutants affect cellular Ca(2+) handling in several ways. We previously have demonstrated that FAD-linked presenilin 2 (PS2) mutants decreas...
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| Main Authors: | , , , , , |
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| Formáid: | Artigo |
| Teanga: | Inglês |
| Foilsithe: |
National Academy of Sciences
2011
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| Ábhair: | |
| Rochtain Ar Líne: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3041131/ https://ncbi.nlm.nih.gov/pubmed/21285369 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.1100735108 |
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