Myocardial Ischemia Activates an Injurious Innate Immune Signaling via Cardiac Heat Shock Protein 60 and Toll-like Receptor 4

Innate immune response after transient ischemia is the most common cause of myocardial inflammation and may contribute to injury, yet the detailed signaling mechanisms leading to such a response are not well understood. Herein we tested the hypothesis that myocardial ischemia activates interleukin r...

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Detalhes bibliográficos
Main Authors: Li, Yan, Si, Rui, Feng, Yan, Chen, Howard H., Zou, Lin, Wang, E., Zhang, Ming, Warren, H. Shaw, Sosnovik, David E., Chao, Wei
Formato: Artigo
Idioma:Inglês
Publicado em: American Society for Biochemistry and Molecular Biology 2011
Assuntos:
Molecular Bases of Disease
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3173057/
https://ncbi.nlm.nih.gov/pubmed/21775438
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M111.246124
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