Coactivation of GR and NFKB alters the repertoire of their binding sites and target genes

Glucocorticoid receptor (GR) exerts anti-inflammatory action in part by antagonizing proinflammatory transcription factors such as the nuclear factor kappa-b (NFKB). Here, we assess the crosstalk of activated GR and RELA (p65, major NFKB component) by global identification of their binding sites and...

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Detalhes bibliográficos
Main Authors: Rao, Nagesha A.S., McCalman, Melysia T., Moulos, Panagiotis, Francoijs, Kees-Jan, Chatziioannou, Aristotelis, Kolisis, Fragiskos N., Alexis, Michael N., Mitsiou, Dimitra J., Stunnenberg, Hendrik G.
Formato: Artigo
Idioma:Inglês
Publicado em: Cold Spring Harbor Laboratory Press 2011
Assuntos:
Research
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3166826/
https://ncbi.nlm.nih.gov/pubmed/21750107
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1101/gr.118042.110
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