A mechanism of suppression of TGF–β/SMAD signaling by NF-κB/RelA

A number of pathogenic and proinflammatory stimuli, and the transforming growth factor-β (TGF-β) exert opposing activities in cellular and immune responses. Here we show that the RelA subunit of nuclear factor κB (NF-κB/RelA) is necessary for the inhibition of TGF-β-induced phosphorylation, nuclear...

Ful tanımlama

Kaydedildi:
Detaylı Bibliyografya
Asıl Yazarlar: Bitzer, Markus, von Gersdorff, Gero, Liang, Dan, Dominguez-Rosales, Alfredo, Beg, Amer A., Rojkind, Marcos, Böttinger, Erwin P.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: Cold Spring Harbor Laboratory Press 2000
Konular:
Research Paper
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC316349/
https://ncbi.nlm.nih.gov/pubmed/10652273
Etiketler: Etiketle
Etiket eklenmemiş, İlk siz ekleyin!

Benzer Materyaller