Specific targeting of the GABA-A receptor α5 subtype by a selective inverse agonist restores cognitive deficits in Down syndrome mice

Antzeko izenburuak

• Chronic Treatment with a Promnesiant GABA-A -Selective Inverse Agonist Increases Immediate Early Genes Expression during Memory Processing in Mice and Rectifies Their Expression Levels in a Down Syndrome Mouse Model
  nork: J. Braudeau, et al.
  Argitaratua: (2011-01-01)
• Chronic Treatment with a Promnesiant GABA-A α5-Selective Inverse Agonist Increases Immediate Early Genes Expression during Memory Processing in Mice and Rectifies Their Expression Levels in a Down Syndrome Mouse Model
  nork: Braudeau, J., et al.
  Argitaratua: (2011)
• Long‐lasting correction of in vivo LTP and cognitive deficits of mice modelling Down syndrome with an α5‐selective GABA(A) inverse agonist
  nork: Duchon, Arnaud, et al.
  Argitaratua: (2020)
• Effects of an α(5)GABA(A) inverse agonist on MK-801-induced learning deficits in an incremental repeated acquisition task
  nork: Povroznik, Jessica M., et al.
  Argitaratua: (2014)
• Dosage of the Abcg1-U2af1 Region Modifies Locomotor and Cognitive Deficits Observed in the Tc1 Mouse Model of Down Syndrome
  nork: Marechal, Damien, et al.
  Argitaratua: (2015)