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Plasmodium falciparum uses a key functional site in complement receptor type-1 for invasion of human erythrocytes
The Plasmodium falciparum adhesin PfRh4 binds to complement receptor type-1 (CR1) on human erythrocytes and mediates a glycophorin-independent invasion pathway. CR1 is a complement regulator and immune-adherence receptor on erythrocytes required for shuttling of C3b/C4b-opsonized particles to liver...
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| Asıl Yazarlar: | , , , , , , , , |
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| Materyal Türü: | Artigo |
| Dil: | Inglês |
| Baskı/Yayın Bilgisi: |
American Society of Hematology
2011
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| Konular: | |
| Online Erişim: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3158720/ https://ncbi.nlm.nih.gov/pubmed/21685372 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1182/blood-2011-03-341305 |
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