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Renal inflammation and elevated blood pressure in a mouse model of reduced β-ENaC
Previous studies suggest β-epithelial Na(+) channel protein (β-ENaC) may mediate myogenic constriction, a mechanism of blood flow autoregulation. A recent study demonstrated that mice with reduced levels of β-ENaC (β-ENaC m/m) have delayed correction of whole kidney blood flow responses, suggesting...
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| Main Authors: | , , , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado: |
American Physiological Society
2011
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| Assuntos: | |
| Acceso en liña: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3154591/ https://ncbi.nlm.nih.gov/pubmed/21543417 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajprenal.00694.2010 |
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