Upregulation of the Ca(V) 1.1-ryanodine receptor complex in a rat model of critical illness myopathy

The processes that trigger severe muscle atrophy and loss of myosin in critical illness myopathy (CIM) are poorly understood. It has been reported that muscle disuse alters Ca(2+) handling by the sarcoplasmic reticulum. Since inactivity is an important contributor to CIM, this finding raises the pos...

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Main Authors: Kraner, Susan D., Wang, Qingbo, Novak, Kevin R., Cheng, Dongmei, Cool, David R., Peng, Junmin, Rich, Mark M.
Formato: Artigo
Idioma:Inglês
Publicado em: American Physiological Society 2011
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Articles
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3119150/
https://ncbi.nlm.nih.gov/pubmed/21474431
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpregu.00032.2011
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