c-Met signaling induces a reprogramming network and supports the glioblastoma stem-like phenotype

The tyrosine kinase c-Met promotes the formation and malignant progression of multiple cancers. It is well known that c-Met hyperactivation increases tumorigenicity and tumor cell resistance to DNA damaging agents, properties associated with tumor-initiating stem cells. However, a link between c-Met...

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Detalhes bibliográficos
Main Authors: Li, Yunqing, Li, Angela, Glas, Martin, Lal, Bachchu, Ying, Mingyao, Sang, Yingying, Xia, Shuli, Trageser, Daniel, Guerrero-Cázares, Hugo, Eberhart, Charles G., Quiñones-Hinojosa, Alfredo, Scheffler, Bjorn, Laterra, John
Formato: Artigo
Idioma:Inglês
Publicado em: National Academy of Sciences 2011
Assuntos:
Biological Sciences
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3116406/
https://ncbi.nlm.nih.gov/pubmed/21628563
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.1016912108
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