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Strain-specific vulnerability to alcohol exposure in utero via hippocampal parent-of-origin expression of deiodinase-III

Prenatal exposure to alcohol is thought to be the most prevalent nongenetic cause of a wide range of neurodevelopmental deficits. Insufficient thyroid hormone levels are one mechanism that hampers development of the alcohol-exposed brain, and we hypothesized that altered dosage of the imprinted thyr...

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Autori principali: Sittig, Laura J., Shukla, Pradeep K., Herzing, Laura B. K., Redei, Eva E.
Natura: Artigo
Lingua:Inglês
Pubblicazione: Federation of American Societies for Experimental Biology 2011
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Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC3114527/
https://ncbi.nlm.nih.gov/pubmed/21429942
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1096/fj.10-179234
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