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Presynaptic silencing is an endogenous neuroprotectant during excitotoxic insults
Glutamate release is a root cause of acute and delayed neuronal damage in response to hypoxic/ischemic insults. Nevertheless, therapeutics that target the postsynaptic compartment have been disappointing clinically. Here we explored whether presynaptic silencing (muting) of glutamatergic terminals i...
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| Autori principali: | , , , |
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| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
2011
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3114267/ https://ncbi.nlm.nih.gov/pubmed/21605675 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.nbd.2011.05.005 |
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