CCR9-CCL25 interactions promote cisplatin resistance in breast cancer cell through Akt activation in a PI3K-dependent and FAK-independent fashion

BACKGROUND: Chemotherapy heavily relies on apoptosis to kill breast cancer (BrCa) cells. Many breast tumors respond to chemotherapy, but cells that survive this initial response gain resistance to subsequent treatments. This leads to aggressive cell variants with an enhanced ability to migrate, inva...

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Bibliografiska uppgifter
Huvudupphovsmän: Johnson-Holiday, Crystal, Singh, Rajesh, Johnson, Erica L, Grizzle, William E, Lillard, James W, Singh, Shailesh
Materialtyp: Artigo
Språk:Inglês
Publicerad: BioMed Central 2011
Ämnen:
Research
Länkar:https://ncbi.nlm.nih.gov/pmc/articles/PMC3110128/
https://ncbi.nlm.nih.gov/pubmed/21539750
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/1477-7819-9-46
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