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Activation of aryl hydrocarbon receptor induces vascular inflammation and promotes atherosclerosis in ApoE−/− mice

OBJECTIVE: Exposure to dioxins has been shown to contribute to the development of inflammatory diseases such as atherosclerosis. Macrophage-mediated inflammation is a critical event in the initiation of atherosclerosis. Previously we showed that treatment of macrophages with 2,3,7,8-tetrachlorodiben...

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Detalhes bibliográficos
Main Authors: Wu, Dalei, Nishimura, Noriko, Kuo, Victoria, Fiehn, Oliver, Shahbaz, Sevini, Winkle, Laura Van, Matsumura, Fumio, Vogel, Christoph
Formato: Artigo
Idioma:Inglês
Publicado em: 2011
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Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3098318/
https://ncbi.nlm.nih.gov/pubmed/21441140
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/ATVBAHA.110.220202
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