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Btn3 is a negative regulator of Btn2-mediated endosomal protein trafficking and prion curing in yeast

Yeast Btn2 facilitates the retrieval of specific proteins from late endosomes (LEs) to the Golgi, a process that may be adversely affected in Batten disease patients. We isolated the putative yeast orthologue of a human complex I deficiency gene, designated here as BTN3, as encoding a Btn2-interacti...

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Bibliografiske detaljer
Main Authors: Kanneganti, Vydehi, Kama, Rachel, Gerst, Jeffrey E.
Format: Artigo
Sprog:Inglês
Udgivet: The American Society for Cell Biology 2011
Fag:
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC3093318/
https://ncbi.nlm.nih.gov/pubmed/21441304
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1091/mbc.E10-11-0878
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