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Transgenic Rescue of Enamel Phenotype in Ambn Null Mice
Ameloblastin null mice fail to make an enamel layer, but the defects could be due to an absence of functional ameloblastin or to the secretion of a potentially toxic mutant ameloblastin. We hypothesized that the enamel phenotype could be rescued by the transgenic expression of normal ameloblastin in...
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| Autori principali: | , , , , , , |
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| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
SAGE Publications
2010
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3085845/ https://ncbi.nlm.nih.gov/pubmed/20940352 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1177/0022034510379223 |
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