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A novel bipartite splicing enhancer modulates the differential processing of the human fibronectin EDA exon.

EDA is a facultative type III homology of human fibronectin encoded by an alternative spliced exon. The EDA+ and EDA- mRNA forms show a cell type specific distribution with their relative proportion varying during development, aging and oncogenic transformation. We have previously demonstrated that...

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Detalhes bibliográficos
Main Authors: Caputi, M, Casari, G, Guenzi, S, Tagliabue, R, Sidoli, A, Melo, C A, Baralle, F E
Formato: Artigo
Idioma:Inglês
Publicado em: 1994
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC307924/
https://ncbi.nlm.nih.gov/pubmed/8152907
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