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A novel bipartite splicing enhancer modulates the differential processing of the human fibronectin EDA exon.
EDA is a facultative type III homology of human fibronectin encoded by an alternative spliced exon. The EDA+ and EDA- mRNA forms show a cell type specific distribution with their relative proportion varying during development, aging and oncogenic transformation. We have previously demonstrated that...
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| 主要な著者: | , , , , , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
1994
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| オンライン・アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC307924/ https://ncbi.nlm.nih.gov/pubmed/8152907 |
| タグ: |
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