Cerebral formation of free radicals during hypoxia does not cause structural damage and is associated with a reduction in mitochondrial PO(2); evidence of O(2)-sensing in humans?

Cellular hypoxia triggers a homeostatic increase in mitochondrial free radical signaling. In this study, blood was obtained from the radial artery and jugular venous bulb in 10 men during normoxia and 9 hours hypoxia (12.9% O(2)). Mitochondrial oxygen tension ([Image: see text]) was derived from cer...

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Detalhes bibliográficos
Main Authors: Bailey, Damian M, Taudorf, Sarah, Berg, Ronan M G, Lundby, Carsten, Pedersen, Bente K, Rasmussen, Peter, Møller, Kirsten
Formato: Artigo
Idioma:Inglês
Publicado em: Nature Publishing Group 2011
Assuntos:
Brief Communication
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3070986/
https://ncbi.nlm.nih.gov/pubmed/21304557
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/jcbfm.2011.2
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