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Disease-associated N-terminal Complement Factor H Mutations Perturb Cofactor and Decay-accelerating Activities
Many mutations associated with atypical hemolytic uremic syndrome (aHUS) lie within complement control protein modules 19–20 at the C terminus of the complement regulator factor H (FH). This region mediates preferential action of FH on self, as opposed to foreign, membranes and surfaces. Hence, spec...
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| Asıl Yazarlar: | , , , , |
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| Materyal Türü: | Artigo |
| Dil: | Inglês |
| Baskı/Yayın Bilgisi: |
American Society for Biochemistry and Molecular Biology
2011
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| Konular: | |
| Online Erişim: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3064162/ https://ncbi.nlm.nih.gov/pubmed/21270465 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M110.211839 |
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