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Loss of an apurinic/apyrimidinic site endonuclease increases the mutagenicity of N-methyl-N'-nitro-N-nitrosoguanidine to Escherichia coli.

xthA- Escherichia coli, which are missing a major cellular apurinic/apyrimidinic (AP) endonuclease, are 5- to 10-fold more sensitive than xthA+ bacteria to mutagenesis by N-methyl-N'-nitro-N-nitrosoguanidine (MNNG) under conditions that induce the "adaptive response." The xthA(-)-dependent mutations...

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Udgivet i:Proc Natl Acad Sci U S A
Principais autores: Foster, P L, Davis, E F
Format: Artigo
Sprog:Inglês
Udgivet: National Academy of Sciences 1987
Fag:
Research Article
Online adgang:https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC304766/
https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/2437587/
https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.84.9.2891
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