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Novel Complement Inhibitor Limits Severity of Experimentally Myasthenia Gravis
OBJECTIVE: Complement mediated injury of the neuromuscular junction is considered a primary disease mechanism in human myasthenia gravis and animal models of experimentally acquired myasthenia gravis (EAMG). We utilized active and passive models of EAMG to investigate the efficacy of a novel C5 comp...
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| Autors principals: | , , , , , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
2009
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3045826/ https://ncbi.nlm.nih.gov/pubmed/19194881 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/ana.21536 |
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