Novel mechanism of increased Ca(2+) release following oxidative stress in neuronal cells involves type 2 inositol-1,4,5-trisphosphate receptors

Dysregulation of Ca(2+) signaling following oxidative stress is an important pathophysiological mechanism of many chronic neurodegenerative disorders, including Alzheimer’s Disease, age-related macular degeneration, glaucomatous and diabetic retinopathies. However, the underlying mechanisms of distu...

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Hlavní autoři: Kaja, Simon, Duncan, R. Scott, Longoria, Sandra, Hilgenberg, Jill D., Payne, Andrew J., Desai, Neeti M., Parikh, Ruby A., Burroughs, Stephanie L., Gregg, Elaine V., Goad, Daryl L., Koulen, Peter
Médium: Artigo
Jazyk:Inglês
Vydáno: 2011
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On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC3038464/
https://ncbi.nlm.nih.gov/pubmed/21075175
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.neuroscience.2010.11.010
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