Complement induces a transient increase in membrane permeability in unlysed erythrocytes.

Lignende værker

• Ca2+-activated K+ efflux limits complement-mediated lysis of human erythrocytes.
  af: Halperin, J A, et al.
  Udgivet: (1989)
• Ligation of complement receptor 1 increases erythrocyte membrane deformability
  af: Glodek, Aleksandra M., et al.
  Udgivet: (2010)
• Detection of complement components on unlysed erythrocytes from acid hemolysis and thrombin test reactions in paroxysmal nocturnal hemoglobinuria.
  af: Jenkins, D E, et al.
  Udgivet: (1966)
• Enhanced complement-mediated lysis of type III paroxysmal nocturnal hemoglobinuria erythrocytes involves increased C9 binding and polymerization.
  af: Hu, V W, et al.
  Udgivet: (1985)
• The transient pore formed by homologous terminal complement complexes functions as a bidirectional route for the transport of autocrine and paracrine signals across human cell membranes.
  af: Acosta, J. A., et al.
  Udgivet: (1996)