Pertussis toxin activates platelets through an interaction with platelet glycoprotein Ib.

Platelets present a unique model to study the B-oligomer effects of pertussis toxin because they become activated in response to the B oligomer but are not susceptible to ADP-ribosylation by the holotoxin. In these studies, the B oligomer of pertussis toxin caused concentration-dependent platelet ac...

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Bibliografiske detaljer
Main Authors: Sindt, K A, Hewlett, E L, Redpath, G T, Rappuoli, R, Gray, L S, Vandenberg, S R
Format: Artigo
Sprog:Inglês
Udgivet: 1994
Fag:
Research Article
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC302934/
https://ncbi.nlm.nih.gov/pubmed/8039878
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