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Prion amyloid structure explains templating: how proteins can be genes
The yeast and fungal prions determine heritable and infectious traits, and are thus genes composed of protein. Most prions are inactive forms of a normal protein as it forms a self-propagating filamentous β – sheet - rich polymer structure called amyloid. Remarkably, a single prion protein sequence...
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| Hoofdauteurs: | , , , , , , , |
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| Formaat: | Artigo |
| Taal: | Inglês |
| Gepubliceerd in: |
2010
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| Onderwerpen: | |
| Online toegang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3025496/ https://ncbi.nlm.nih.gov/pubmed/20726897 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/j.1567-1364.2010.00666.x |
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