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Soleus muscle in glycosylation-deficient muscular dystrophy is protected from contraction-induced injury
The glycosylation of dystroglycan is required for its function as a high-affinity laminin receptor, and loss of dystroglycan glycosylation results in congenital muscular dystrophy. The purpose of this study was to investigate the functional defects in slow- and fast-twitch muscles of glycosylation-d...
Gorde:
| Egile Nagusiak: | , , , , |
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| Formatua: | Artigo |
| Hizkuntza: | Inglês |
| Argitaratua: |
American Physiological Society
2010
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| Gaiak: | |
| Sarrera elektronikoa: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3006333/ https://ncbi.nlm.nih.gov/pubmed/20844247 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpcell.00192.2010 |
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