Thyroid hormone inhibits ERK phosphorylation in pressure overload-induced hypertrophied mouse hearts through a receptor-mediated mechanism

Pressure overload-induced cardiac hypertrophy results in a pathological type of hypertrophy with activation of signaling cascades like the extracellular signal-regulated kinase (ERK) pathway, which promotes negative cardiac remodeling and decreased contractile function. In contrast, thyroid hormone...

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Bibliographische Detailangaben
Hauptverfasser: Suarez, Jorge, Scott, Brian T., Suarez-Ramirez, Jorge A., Chavira, Citlalic V., Dillmann, Wolfgang H.
Format: Artigo
Sprache:Inglês
Veröffentlicht: American Physiological Society 2010
Schlagworte:
Receptors and Signal Transduction
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC3006330/
https://ncbi.nlm.nih.gov/pubmed/20926779
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpcell.00168.2010
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