Ischemic Preconditioning Decreases Mitochondrial Proton Leak and Reactive Oxygen Species Production in the Postischemic Heart

BACKGROUND: Proton leak (H(+) leak) dissipates mitochondrial membrane potential (mΔΨ) through the reentry of protons into the mitochondrial matrix independent of ATP synthase. Changes in H(+) leak may affect reactive oxygen species (ROS) production. We measured H(+) leak and ROS production during is...

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Hlavní autoři: Quarrie, Ricardo, Cramer, Brandon M., Lee, Daniel S., Steinbaugh, Gregory E., Erdahl, Warren, Pfeiffer, Douglas R., Zweier, Jay L., Crestanello, Juan A.
Médium: Artigo
Jazyk:Inglês
Vydáno: 2010
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On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC3005326/
https://ncbi.nlm.nih.gov/pubmed/21035133
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.jss.2010.09.018
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