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Excitotoxicity through Ca(2+)-permeable AMPA receptors requires Ca(2+)-dependent JNK activation
The GluA4-containing Ca(2+)-permeable α-amino-3-hydroxy-5-methyl-4-isoxazole-propionic acid receptors (Ca-AMPARs) were previously shown to mediate excitotoxicity through mechanisms involving the activator protein-1 (AP-1), a c-Jun N-terminal kinase (JNK) substrate. To further investigate JNK involve...
Kaydedildi:
| Asıl Yazarlar: | , , , , , , , |
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| Materyal Türü: | Artigo |
| Dil: | Inglês |
| Baskı/Yayın Bilgisi: |
2010
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| Konular: | |
| Online Erişim: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3003258/ https://ncbi.nlm.nih.gov/pubmed/20708684 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.nbd.2010.08.008 |
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