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Rab8A and Rab13 are activated by insulin and regulate GLUT4 translocation in muscle cells

Skeletal muscle is the primary site of dietary glucose disposal, a function that depends on insulin-mediated exocytosis of GLUT4 vesicles to its cell surface. In skeletal muscle and adipocytes, this response involves Akt signaling to the Rab-GAP (GTPase-activating protein) AS160/TBC1D4. Intriguingly...

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Autores principales: Sun, Yi, Bilan, Philip J., Liu, Zhi, Klip, Amira
Formato: Artigo
Lenguaje:Inglês
Publicado: National Academy of Sciences 2010
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Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC2993354/
https://ncbi.nlm.nih.gov/pubmed/21041651
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.1009523107
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