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Rab8A and Rab13 are activated by insulin and regulate GLUT4 translocation in muscle cells
Skeletal muscle is the primary site of dietary glucose disposal, a function that depends on insulin-mediated exocytosis of GLUT4 vesicles to its cell surface. In skeletal muscle and adipocytes, this response involves Akt signaling to the Rab-GAP (GTPase-activating protein) AS160/TBC1D4. Intriguingly...
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| Main Authors: | , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
National Academy of Sciences
2010
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2993354/ https://ncbi.nlm.nih.gov/pubmed/21041651 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.1009523107 |
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