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Cannabinoid-1 (CB1) receptor gene deletion has a compartment-specific affect on the dendritic and axonal availability of mu-opioid receptors and on dopamine axons in the mouse nucleus accumbens
Cannabinoid-type 1 (CB1) receptors are implicated in mu-opioid receptor (mu-OR)-dependent reward ascribed partially to mesolimbic dopamine release in the nucleus accumbens (Acb) shell. Thus, CB1 receptor gene deletion may preferentially alter the availability of mu-ORs and/or dopamine innervation in...
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| Main Authors: | , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2010
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2954666/ https://ncbi.nlm.nih.gov/pubmed/20939059 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/syn.20807 |
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