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Cannabinoid-1 (CB1) receptor gene deletion has a compartment-specific affect on the dendritic and axonal availability of mu-opioid receptors and on dopamine axons in the mouse nucleus accumbens

Cannabinoid-type 1 (CB1) receptors are implicated in mu-opioid receptor (mu-OR)-dependent reward ascribed partially to mesolimbic dopamine release in the nucleus accumbens (Acb) shell. Thus, CB1 receptor gene deletion may preferentially alter the availability of mu-ORs and/or dopamine innervation in...

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Bibliografiske detaljer
Main Authors: Lane, Diane A., Chan, June, Lupica, Carl R., Pickel, Virginia M.
Format: Artigo
Sprog:Inglês
Udgivet: 2010
Fag:
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC2954666/
https://ncbi.nlm.nih.gov/pubmed/20939059
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/syn.20807
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