GLUCOSE-INDUCED INTESTINAL VASODILATION VIA ADENOSINE A1 RECEPTORS REQUIRES NITRIC OXIDE BUT NOT K(+)(ATP) CHANNELS

INTRODUCTION: Both nitric oxide (NO) and adenosine A1 receptor activation mediate microvascular vasodilation during intestinal glucose absorption. Our overall hypothesis is that ATP utilization during glucose absorption would increase adenosine metabolite release, which acts on adenosine A1 receptor...

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Huvudupphovsmän: Matheson, Paul J., Li, Na, Harris, Patrick D., Zakaria, El Rasheid, Garrison, R. Neal
Materialtyp: Artigo
Språk:Inglês
Publicerad: 2010
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Article
Länkar:https://ncbi.nlm.nih.gov/pmc/articles/PMC2953599/
https://ncbi.nlm.nih.gov/pubmed/20452612
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.jss.2010.02.013
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