Mineralocorticoid Accelerates Transition to Heart Failure with Preserved Ejection Fraction Via “Non-Genomic Effects”

BACKGROUND: Mechanisms promoting the transition from hypertensive heart disease (HHD) to heart failure with preserved ejection fraction (HFpEF) are poorly understood. When inappropriate for salt status, mineralocorticoid (deoxycorticosterone acetate, DOCA) excess causes hypertrophy, fibrosis and dia...

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書誌詳細
主要な著者: Mohammed, Selma F., Ohtani, Tomohito, Korinek, Josef, Lam, Carolyn S.P., Larsen, Katarina, Simari, Robert D., Valencik, Maria L., Burnett, John C., Redfield, Margaret M.
フォーマット: Artigo
言語:Inglês
出版事項: 2010
主題:
Article
オンライン･アクセス:https://ncbi.nlm.nih.gov/pmc/articles/PMC2938025/
https://ncbi.nlm.nih.gov/pubmed/20625113
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/CIRCULATIONAHA.109.915215
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