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Laminin deficits induce alterations in the development of dopaminergic neurons in the mouse retina
Genetically modified mice lacking the β2 laminin chain (β2null), the γ3 laminin chain (γ3 null), or both β2/γ3 chains (compound null) were produced. The development of tyrosine hydroxylase (TH) immunoreactive neurons in these mouse lines was studied between birth and postnatal day (P) 20. Compared t...
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| Autors principals: | , , , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
2007
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2935900/ https://ncbi.nlm.nih.gov/pubmed/17711601 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1017/S0952523807070514 |
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