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A novel KCNA5 Mutation Implicates Tyrosine Kinase Signaling in Human Atrial Fibrillation
BACKGROUND: Emerging evidence has strongly implicated hereditary determinants for atrial fibrillation (AF). Loss-of-function mutations in KCNA5 encoding the ultra-rapid delayed rectifier potassium current (I(Kur)) have been identified in AF families. OBJECTIVE: Our study sought to present the clinic...
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| Autors principals: | , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
2010
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2932792/ https://ncbi.nlm.nih.gov/pubmed/20638934 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.hrthm.2010.05.032 |
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