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Fluoxetine protects against amyloid-beta toxicity, in part via daf-16 mediated cell signaling pathway, in Caenorhabditis elegans
Alzheimer’s disease (AD) is a progressive neurodegenerative disorder and is the most common form of dementia in elderly people. The accumulation of amyloid β (Aβ) is one of the histopathological hallmarks of AD. Aβ is aggregated to form oligomers which are toxic to neurons and are critical to the on...
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| Main Authors: | , , |
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| Format: | Artigo |
| Sprog: | Inglês |
| Udgivet: |
2010
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| Fag: | |
| Online adgang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2926186/ https://ncbi.nlm.nih.gov/pubmed/20420844 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.neuropharm.2010.04.008 |
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