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Astrocytic gap junctional communication is reduced in amyloid-β-treated cultured astrocytes, but not in Alzheimer's disease transgenic mice

Alzheimer's disease is characterized by accumulation of amyloid deposits in brain, progressive cognitive deficits and reduced glucose utilization. Many consequences of the disease are attributed to neuronal dysfunction, but roles of astrocytes in its pathogenesis are not well understood. Astroc...

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Λεπτομέρειες βιβλιογραφικής εγγραφής
Κύριοι συγγραφείς: Cruz, Nancy F, Ball, Kelly K, Dienel, Gerald A
Μορφή: Artigo
Γλώσσα:Inglês
Έκδοση: American Society for Neurochemistry 2010
Θέματα:
Διαθέσιμο Online:https://ncbi.nlm.nih.gov/pmc/articles/PMC2922840/
https://ncbi.nlm.nih.gov/pubmed/20730033
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1042/AN20100017
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