Inhibition of c-Jun Kinase Provides Neuroprotection in a Model of Alzheimer’s Disease

The c-Jun N-terminal kinase (JNK) pathway potentially links together the three major pathological hallmarks of Alzheimer’s disease (AD): development of amyloid plaques, neurofibrillary tangles, and brain atrophy. As activation of the JNK pathway has been observed in amyloid models of AD in associati...

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Detalhes bibliográficos
Main Authors: Braithwaite, Steven P., Schmid, Ralf S., He, Dong Ning, Sung, Mei-Li A., Cho, Seongeon, Resnick, Lynn, Monaghan, Michael M., Hirst, Warren D., Essrich, Christian, Reinhart, Peter H., Lo, Donald C.
Formato: Artigo
Idioma:Inglês
Publicado em: 2010
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Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC2910136/
https://ncbi.nlm.nih.gov/pubmed/20451607
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.nbd.2010.04.015
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