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A myopathy-linked tropomyosin mutation severely alters thin filament conformational changes during activation
Human point mutations in β- and γ-tropomyosin induce contractile deregulation, skeletal muscle weakness, and congenital myopathies. The aim of the present study was to elucidate the hitherto unknown underlying molecular mechanisms. Hence, we recorded and analyzed the X-ray diffraction patterns of hu...
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| Hlavní autoři: | , , , |
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| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
National Academy of Sciences
2010
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2906900/ https://ncbi.nlm.nih.gov/pubmed/20457903 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.1001733107 |
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