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Mechanistic links between Na(+) channel (SCN5A) mutations and impaired cardiac pacemaking in sick sinus syndrome

RATIONALE: Familial sick sinus syndrome (SSS) has been linked to loss-of-function mutations of the SCN5A gene, which result in decreased inward Na(+) current, I(Na). However, the functional role of I(Na) in cardiac pacemaking is controversial, and mechanistic links between the mutations and sinus no...

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Bibliografiske detaljer
Main Authors: Butters, Timothy D., Aslanidi, Oleg V., Inada, Shin, Boyett, Mark R., Hancox, Jules C., Lei, Ming, Zhang, Henggui
Format: Artigo
Sprog:Inglês
Udgivet: 2010
Fag:
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC2901593/
https://ncbi.nlm.nih.gov/pubmed/20448214
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/CIRCRESAHA.110.219949
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