Biological effects of CCS in the absence of SOD1 enzyme activation: implications for disease in a mouse model for ALS

Podobne knjige/članki

• Redox susceptibility of SOD1 mutants is associated with the differential response to CCS over-expression in vivo
  od: Son, Marjatta, et al.
  Izdano: (2009)
• Isolated Cytochrome c Oxidase Deficiency in G93A SOD1 Mice Overexpressing CCS Protein
  od: Son, Marjatta, et al.
  Izdano: (2008)
• Overexpression of CCS in G93A-SOD1 mice leads to accelerated neurological deficits with severe mitochondrial pathology
  od: Son, Marjatta, et al.
  Izdano: (2007)
• S-acylation of SOD1, CCS, and a stable SOD1-CCS heterodimer in human spinal cords from ALS and non-ALS subjects
  od: Antinone, Sarah E., et al.
  Izdano: (2017)
• Mechanisms for activating Cu- and Zn-containing superoxide dismutase in the absence of the CCS Cu chaperone
  od: Carroll, Mark C., et al.
  Izdano: (2004)