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Food demand and meal size in mice with single or combined disruption of melanocortin type 3 and 4 receptors

Mice with homozygous genetic disruption of the melanocortin-4 receptor gene (MC4R−/−) are known to be hyperphagic and become obese, while those with disruption of the melanocortin-3 receptor gene (MC3R−/−) do not become markedly obese. The contribution of MC3R signaling in energy homeostasis remains...

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Библиографические подробности
Главные авторы: Atalayer, Deniz, Robertson, Kimberly L., Haskell-Luevano, Carrie, Andreasen, Amy, Rowland, Neil E.
Формат: Artigo
Язык:Inglês
Опубликовано: American Physiological Society 2010
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Online-ссылка:https://ncbi.nlm.nih.gov/pmc/articles/PMC2886701/
https://ncbi.nlm.nih.gov/pubmed/20375267
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpregu.00562.2009
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