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GLUT-2 function in glucose-unresponsive beta cells of dexamethasone-induced diabetes in rats.
Spontaneous and dexamethasone-induced noninsulin-dependent diabetes mellitus (NIDDM) in rats is associated with loss of glucose-stimulated insulin secretion (GSIS) and a reduction in both GLUT-2-positive beta cells and high Km glucose transport. To determine if the chronology and correlation of thes...
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| Vydáno v: | J Clin Invest |
|---|---|
| Hlavní autoři: | , , , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
American Society for Clinical Investigation
1993
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC288361/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/8408647/ https://ncbi.nlm.nih.govhttps://doi.org/10.1172/JCI116788 |
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